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CSE and CBS are downregulated and this may contribute to the elevated oxidative stress associated with the disease (Murano, 1995)

AMPK might play a role in facilitating the movement of FAT/CD36 and FABPpm to the cell surface (129, 130)

For example, a PBPK model for a liver-targeted formulation of a hepatoprotective TCM compound (say silybin) can be calibrated, and then AI can be used to test virtual scenariose.g., What if we use a galactose-targeted nanoparticle to exploit hepatocyte receptors? and quantify the likely increase in liver deposition versus off-target distribution