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doi:10.7326/0003-4819-147-4-200708210-00175 Grtner R, Gasnier BC, Dietrich JW, Krebs B, Angstwurm MW

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As reviewed elsewhere [79, 86], impairments in redox homeostasis and susceptibility of increased oxidative stress are linked to glutamatergic dysfunction due to (1) hypoactive NMDA receptors, (2) to degeneration of fast-spiking parvalbumin-positive GABAergic interneurons that are essential for fast local neuronal synchronization, (3) to dysfunctional oligodendrocytes resulting in poor myelination and thus impairing axonal integrity and signal conduction across brain areas, and (4) to neuroinflammation