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Inhibition of arginase modulates T-cell response in the tumor microenvironment of lung carcinoma

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Although it is conceivable that PC9 and HCC44 may also have some abnormalities that activate NRF2, it may suggest that the presence of proficient SWI/SNF complexes contributes to the expression of SLC7A11 rather than that NRF2 activating mutations are involved in high expression of SLC7A11