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Excessive lipid metabolic reprogramming in OA significantly raises ferroptosis risk, so targeting lipid synthesis, transport, and degradation represents a promising upstream approach to reducing ferroptotic damage

BCL2L1 and BCL2 are anti-apoptotic proteins belonging to the BCL2 family that function by preventing the oligomerization of the BAX and BAK proteins and maintaining the integrity of mitochondrial membrane, thus inhibiting cell apoptosis at high expression 37

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Study Duration The study duration is dependent on the investigated drug and its expected effects